Late-Breaking KERENDIA® (finerenone)
Investigational Data Showed a Statistically Significant Reduction
in the Composite Outcome of Cardiovascular Death and Total Heart
Failure Events in Adult Patients with Heart Failure with Mildly
Reduced or Preserved Ejection Fraction
- The primary endpoint results were consistent across all
prespecified subgroups 1
- KERENDIA is the first-and-only non-steroidal mineralocorticoid
receptor antagonist (MRA) to meet a primary composite
cardiovascular endpoint in a Phase III trial investigating patients
with HF with mildly reduced or preserved ejection fraction (LVEF
≥40%)1
- Results from FINEARTS-HF were simultaneously published in the
New England Journal of Medicine
Detailed results from the Phase III FINEARTS-HF trial presented
today at the European Society of Cardiology (ESC) Congress 2024
during a late-breaking Hot Line session and simultaneously
published in the New England Journal of Medicine showed that
KERENDIA® (finerenone) achieved a statistically significant
reduction of the composite of cardiovascular death and total (first
and recurrent) heart failure (HF) events, defined as either an
unplanned hospitalization for HF or an urgent HF visit, by 16%
(rate ratio, 0.84; 95% confidence interval [CI], 0.74 to 0.95; P =
0.007) in patients with HF and a LVEF ≥40% (left ventricular
ejection fraction) compared to placebo in addition to a patients’
prescribed treatment regimen.1
KERENDIA is the first-and-only non-steroidal mineralocorticoid
receptor antagonist (MRA) to meet a primary composite
cardiovascular endpoint in a Phase III trial investigating patients
with HF and LVEF ≥40%.1
KERENDIA is currently approved to reduce the risk of
cardiovascular death, hospitalization for HF, non-fatal myocardial
infarction (MI), sustained eGFR decline, and end-stage kidney
disease in adult patients with chronic kidney disease (CKD)
associated with type 2 diabetes (T2D).2
KERENDIA already has established cardiovascular benefit
(reduction in hospitalization for HF, CV death and non-fatal MI) in
adults with CKD associated with T2D,2 and this new data provides
positive results in a different patient population not limited to
CKD in T2D – patients diagnosed with HF (LVEF ≥40).1
FINEARTS-HF is a multicenter, randomized, double-blind,
parallel-group, placebo-controlled, event-driven Phase III trial.
It is investigating the efficacy and safety of finerenone for the
reduction of risk of cardiovascular death and HF events in patients
with a diagnosis of symptomatic HF (New York Heart Association
class II-IV) with a LVEF of ≥40%, a measurement that indicates how
much blood the left ventricle of the heart pumps with each beat.3
In the FINEARTS-HF trial, no new safety signals were identified
compared with those seen in previous studies with the
compound.1
“Treating heart failure patients with LVEF ≥40% has provided a
challenge for many physicians, as these patients have a substantial
risk for cardiovascular events. Unlike heart failure with reduced
ejection fraction, there are limited guideline-directed options for
heart failure with LVEF >40%,” said
Scott D. Solomon, MD, The Edward D. Frohlich Distinguished Chair,
Professor of Medicine at Harvard Medical School, Director of
Non-invasive Cardiology and Senior Physician at Brigham and Women’s
Hospital and Chair of the trial’s Executive Committee. “FINEARTS-HF
is the first large-scale trial where a non-steroidal
mineralocorticoid receptor antagonist met a primary composite
cardiovascular endpoint in patients with heart failure with mildly
reduced or preserved ejection fraction (LVEF >40%), and these results provide insights for
the cardiology community managing these high-risk patients.”
“Bayer has a strong heritage in cardiology, and heart failure is
a key focus area for us, with these results underpinning our
ongoing commitment to patients with this devastating condition.
Based on FINEARTS-HF, KERENDIA has shown to reduce the risk of
cardiovascular outcomes in a complex to treat patient population,”
said Dr. Christian Rommel, Head of Research and Development at
Bayer’s Pharmaceuticals Division.
The results shown in the primary endpoint results were
consistent across all prespecified subgroups. Secondary outcomes
tested hierarchically included total number of worsening HF events
(rate ratio, 0.82; 95% confidence interval [CI], 0.71 to 0.94,
p=0.006) and the Kansas City Cardiomyopathy Questionnaire (KCCQ)
Total Symptom Score (TSS) assessed at months six, nine and 12.
There was a mean change from baseline in the KCCQ-TSS of 8 points
in the finerenone group and 6.4 points in the placebo group
(between-group difference of 1.6 points; 95%CI, 0.8 to 2.3,
p<0.001).
Serious adverse events were comparable between treatment groups,
occurring in 38.7% (1,157/2,993) of the finerenone group and 40.5%
(1,213/2,993) of the placebo group. Discontinuation of the trial
drug for reasons other than death was similar between groups, with
20.4% (611/2,993) in the finerenone group and 20.6% (616/2,993) in
the placebo group. Increases in creatinine and potassium levels
were more frequent in patients receiving finerenone compared to
placebo, with investigator-reported hyperkalemia in 9.7%
(289/2,993) of finerenone-treated patients versus 4.2% (125/2,993)
in the placebo group. Serum potassium levels >6 mmol/L were
observed in 3% (n=86) of the finerenone group compared to 1.4%
(41/2,993) in the placebo group. While hyperkalemia was more common
with finerenone, it rarely led to hospitalization (0.5% [16/2,993]
versus 0.2% [6/2,993] in the placebo group), and no cases resulted
in death.1
HF is a complex clinical syndrome with symptoms and signs that
result from any structural or functional impairment of ventricular
filling or ejection of blood.4 Approximately 6.7 million adults in
the U.S. live with HF, of whom about 55% have a LVEF ≥40%.5 Despite
the high prevalence, guideline-directed medical treatment options
for patients with HF with LVEF ≥40% are limited.6 This patient
group is often balancing multiple comorbidities, such as obesity,
hypertension and CKD.4
Bayer plans to discuss this data and submission for regulatory
approval with the U.S. Food and Drug Administration (FDA).
About FINEARTS-HF3
FINEARTS-HF is a randomized, double-blind, placebo-controlled,
multicenter, event-driven Phase III trial investigating the
efficacy and safety of KERENDIA® (finerenone) for the reduction of
risk of cardiovascular death and heart failure (HF) events in
patients with a diagnosis of symptomatic heart failure (New York
Heart Association class II-IV) with a left ventricular ejection
fraction (LVEF) of ≥40%, measured by local imaging measurement
within the last 12 months, who received diuretic treatment for at
least 30 days prior to randomization. The primary endpoint of
FINEARTS-HF was the composite of cardiovascular death and total
(first and recurrent) HF events, defined as hospitalizations for HF
or urgent HF visits.
Approximately 6,000 patients were randomized to receive KERENDIA
or placebo once daily for up to 42 months. Results showed that
KERENDIA once daily in addition to a patients’ prescribed treatment
regimen reduced the composite of total worsening HF events and
cardiovascular death in patients with HF and mildly reduced or
preserved ejection fraction. KERENDIA also significantly reduced
the secondary endpoints of total HF events (HR 0.82 [95% CI,
0.71-0.94; p=0.0062]) and improved patient-reported health status
as measured by the change from baseline in Total Symptom Score of
Kansas City Cardiomyopathy Questionnaire (KCCQ-TSS) (between-group
difference 1.6 points [95% CI, 0.8-2.3; p<0.0001]).
With overall more than 15,000 patients, the MOONRAKER clinical
trial program with KERENDIA, including FINEARTS-HF, is one of the
largest HF trial programs to date and aims to establish a
comprehensive body of evidence for KERENDIA across a broad spectrum
of patients and clinical settings.1
About KERENDIA® (finerenone)2
KERENDIA is a non-steroidal mineralocorticoid receptor
antagonist (MRA) and was approved by the U.S. Food and Drug
Administration (FDA) in July 2021 to reduce the risk of sustained
eGFR decline, end-stage kidney disease, cardiovascular death,
non-fatal myocardial infarction, and hospitalization for HF in
adults with CKD associated with T2D.
In adults with CKD associated with T2D, KERENDIA has been
recommended to reduce the risk of hospitalization for HF by the
American Diabetes Association (ADA)7 and European Society of
Cardiology (ESC).8
IMPORTANT SAFETY INFORMATION
CONTRAINDICATIONS:
- Concomitant use with strong CYP3A4 inhibitors
- Patients with adrenal insufficiency
WARNINGS AND PRECAUTIONS:
- Hyperkalemia: KERENDIA can cause hyperkalemia. The risk
for developing hyperkalemia increases with decreasing kidney
function and is greater in patients with higher baseline potassium
levels or other risk factors for hyperkalemia. Measure serum
potassium and eGFR in all patients before initiation of treatment
with KERENDIA and dose accordingly. Do not initiate KERENDIA if
serum potassium is >5.0 mEq/L. Measure serum potassium
periodically during treatment with KERENDIA and adjust dose
accordingly. More frequent monitoring may be necessary for patients
at risk for hyperkalemia, including those on concomitant
medications that impair potassium excretion or increase serum
potassium.
MOST COMMON ADVERSE REACTIONS:
- From the pooled data of 2 placebo-controlled studies, the
adverse reactions reported in ≥1% of patients on KERENDIA and more
frequently than placebo were hyperkalemia (14% versus 6.9%),
hypotension (4.6% versus 3.9%), and hyponatremia (1.3% versus
0.7%).
DRUG INTERACTIONS:
- Strong CYP3A4 Inhibitors: Concomitant use of KERENDIA
with strong CYP3A4 inhibitors is contraindicated. Avoid concomitant
intake of grapefruit or grapefruit juice.
- Moderate and Weak CYP3A4 Inhibitors: Monitor serum
potassium during drug initiation or dosage adjustment of either
KERENDIA or the moderate or weak CYP3A4 inhibitor and adjust
KERENDIA dosage as appropriate.
- Strong and Moderate CYP3A4 Inducers: Avoid concomitant
use of KERENDIA with strong or moderate CYP3A4 inducers.
USE IN SPECIFIC POPULATIONS:
- Lactation: Avoid breastfeeding during treatment with
KERENDIA and for 1 day after treatment.
- Hepatic Impairment: Avoid use of KERENDIA in patients
with severe hepatic impairment (Child Pugh C) and consider
additional serum potassium monitoring with moderate hepatic
impairment (Child Pugh B).
Please click here for full Prescribing
Information for KERENDIA.
About Bayer’s Commitment in Cardiovascular and Kidney
Diseases
A leader in the cardiovascular (CV) space, Bayer upholds a
long-standing commitment to delivering science for a better life by
advancing research and treatment options. Bayer’s cardiorenal
franchise, which began with the discovery and development of a
number of vital therapies, now includes several products and
compounds in various stages of preclinical and clinical development
with the potential to impact the way that CV and kidney diseases
are treated.
Bayer is investigating potential treatment approaches for areas
of high unmet medical need. Currently, Bayer is investigating nine
CVR-related projects in different stages of development, including
heart failure (HF) and non-diabetic chronic kidney disease
(CKD).9
Bayer is actively identifying resources and programs aimed at
better understanding the real-world management of CKD, expanding
screening and early care management for CKD, aligning with and
supporting groups and institutions that share the common goals of
improving health outcomes, promoting health literacy and education,
and promoting research and initiatives that represent the diversity
required to address the needs of all patients.
About Bayer
Bayer is a global enterprise with core competencies in the life
science fields of health care and nutrition. In line with its
mission, “Health for all, Hunger for none,” the company’s products
and services are designed to help people and the planet thrive by
supporting efforts to master the major challenges presented by a
growing and aging global population. Bayer is committed to driving
sustainable development and generating a positive impact with its
businesses. At the same time, the Group aims to increase its
earning power and create value through innovation and growth. The
Bayer brand stands for trust, reliability and quality throughout
the world. In fiscal 2023, the Group employed approximately 100,000
people and had sales of 47.6 billion euros. R&D expenses before
special items amounted to 5.8 billion euros. For more information,
go to www.bayer.com.
Find more information at https://pharma.bayer.com/ Follow us on
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Forward-Looking Statements
This release may contain forward-looking statements based on
current assumptions and forecasts made by Bayer management. Various
known and unknown risks, uncertainties and other factors could lead
to material differences between the actual future results,
financial situation, development or performance of the company and
the estimates given here. These factors include those discussed in
Bayer’s public reports, which are available on the Bayer website at
www.bayer.com. The company assumes no liability whatsoever to
update these forward-looking statements or to conform them to
future events or developments.
____________________________
1 Data on file. 2 Bayer Pharmaceuticals. Kerendia (finerenone)
[package insert]. U.S. Food and Drug Administration. Available at:
https://www.accessdata.fda.gov/drugsatfda_docs/label/2021/215341s000lbl.pdf.
Accessed August 30, 2024. 3 Study to Evaluate the Efficacy (Effect
on Disease) and Safety of Finerenone on Morbidity (Events
Indicating Disease Worsening) & Mortality (Death Rate) in
Participants With Heart Failure and Left Ventricular Ejection
Fraction (Proportion of Blood Expelled Per Heart Stroke) Greater or
Equal to 40% (FINEARTS-HF). Clinical trial registration No.
NCT04435626. https://clinicaltrials.gov/study/NCT04435626. Accessed
August 30, 2024. 4 Heidenreich P, et al. 2022 AHA/ACC/HFSA
Guideline for the Management of Heart Failure: A Report of the
American College of Cardiology/American Heart Association Joint
Committee on Clinical Practice Guidelines. J Am Coll Cardiol. 2023.
https://pubmed.ncbi.nlm.nih.gov/35379503/. Accessed August 30,
2024. 5 Bozkurt A, et al. Heart Failure Epidemiology and Outcomes
Statistics: A Report of the Heart Failure Society of America. J
Card Fail. 2023; Oct;29(10):1412-1451. doi:
10.1016/j.cardfail.2023.07.006. Epub 2023 Sep 26. PMID: 37797885;
PMCID: PMC10864030. 6 Desai N, et al. Heart failure with mildly
reduced and preserved ejection fraction: A review of disease burden
and remaining unmet medical needs within a new treatment landscape.
Heart Fail Rev. 2024;29(3):631-662.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11035416/. Accessed
August 30, 2024. 7 American Diabetes Association (Section 10:
Cardiovascular disease and risk management: standards of care in
diabetes—2024.). Diabetes Care. 2024;47(Suppl. 1):S179-S218.
doi:10.2337/dc24-S010. 8 Marx N, et al. ESC Guidelines for the
management of cardiovascular disease in patients with diabetes. Eur
Heart J. 2023;44(39):4043-4140. doi:10.1093/eurheartj/ehad192. 9
Bayer Pharmaceuticals. Development Pipeline. U.S.
https://www.bayer.com/en/pharma/development-pipeline. Accessed
August 30, 2024.
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Media Contact: Elaine Colón
Bayer Media Relations Elaine.colon@bayer.com +1-732-236-1587